Dann erzählt mir mal, warum ein Diabetiker Coke Zero ohne Probleme trinken kann #popcorn
Würde eher darauf tippen, dass die großen Mengen Phosphorsäure dem Magen nicht so gefallen, aber mit deren Verstoffwechselung kenn ich mich nicht aus
Jo sicher. Aber hab im Inet was gelesen, dass es Feuchtigkeit aus der Luft ziehen kann und da es hier etwas um 40° ist und es eben einige Stunden dauert, bis ich im kühlen Deutschland bin, bin ich mir nicht sichert.
Jo sicher. Aber hab im Inet was gelesen, dass es Feuchtigkeit aus der Luft ziehen kann und da es hier etwas um 40° ist und es eben einige Stunden dauert, bis ich im kühlen Deutschland bin, bin ich mir nicht sichert.
Meinste, das geht klar?!
Also wenn ich im Ausland meine Koks Bestellungen abhole, kühle ich die auch nie.....
Das past wirklich was ihr vermutet, wenn ich trinke dann esse ich meistens nichts dazu und fühle mich dann so schlapp...
Für nen BB hört sich das mit Insulin, hypoglykämisch nicht gerade gut an oder?
Werd jetzt mal bewußt testen, Coke mit und ohne Essen und berichten wenns euch interessiert...
Danke
So als Idee: könnte die Schlappheit nicht auch an einer Koffeingewöhnung liegen ?
So als Idee: könnte die Schlappheit nicht auch an einer Koffeingewöhnung liegen ?
Weiß ich nicht!
Ich bin mir auch nicht sicher ob in CokeZero wirklich Koffein drinne ist! Es steht zwar drauf koffeinhaltiges Getränk, bei der Auflistung der Inhaltsstoffe allerdings Aroma Koffein...
Allgemein kann ich noch sagen das ich seit 4 Monaten CokeZero anstelle normaler Cola trinke...Nicht täglich, aber wenn, dann Kill ich die Flasche relativ zügig...
Mein Allgemeiner Koffein Konsum in Form von Kaffee ist seit Anfang Juli zurück gegangen, seitdem ich am Ernährungsplan basteln bin... Morgens ne Tasse, und noch Mittags oder Nachmittags. Früher hab ich jeweils 2 Tassen getrunken...
Butchko HH, Stargel WW, Comer CP, Mayhew DA, Benninger C, Blackburn GL, de Sonneville LM, Geha RS, Hertelendy Z, Koestner A, Leon AS, Liepa GU, McMartin KE, Mendenhall CL, Munro IC, Novotny EJ, Renwick AG, Schiffman SS, Schomer DL, Shaywitz BA, Spiers PA, Tephly TR, Thomas JA, Trefz FK.
Medical and Scientific Affairs, The NutraSweet Company, Mt Prospect, Illinois 60056, USA. harriett.h.butchko@nutrasweet.com
Abstract
Over 20 years have elapsed since aspartame was approved by regulatory agencies as a sweetener and flavor enhancer. The safety of aspartame and its metabolic constituents was established through extensive toxicology studies in laboratory animals, using much greater doses than people could possibly consume. Its safety was further confirmed through studies in several human subpopulations, including healthy infants, children, adolescents, and adults; obese individuals; diabetics; lactating women; and individuals heterozygous (PKUH) for the genetic disease phenylketonuria (PKU) who have a decreased ability to metabolize the essential amino acid, phenylalanine. Several scientific issues continued to be raised after approval, largely as a concern for theoretical toxicity from its metabolic components--the amino acids, aspartate and phenylalanine, and methanol--even though dietary exposure to these components is much greater than from aspartame. Nonetheless, additional research, including evaluations of possible associations between aspartame and headaches, seizures, behavior, cognition, and mood as well as allergic-type reactions and use by potentially sensitive subpopulations, has continued after approval. These findings are reviewed here. The safety testing of aspartame has gone well beyond that required to evaluate the safety of a food additive. When all the research on aspartame, including evaluations in both the premarketing and postmarketing periods, is examined as a whole, it is clear that aspartame is safe, and there are no unresolved questions regarding its safety under conditions of intended use.
The potential toxicity of artificial sweeteners.
Whitehouse CR, Boullata J, McCauley LA.
Adult Health/Gerontology Nurse Practitioner Program, School of Nursing, University of Pennsylvania, Philadelphia, PA, USA.
Abstract
Since their discovery, the safety of artificial sweeteners has been controversial. Artificial sweeteners provide the sweetness of sugar without the calories. As public health attention has turned to reversing the obesity epidemic in the United States, more individuals of all ages are choosing to use these products. These choices may be beneficial for those who cannot tolerate sugar in their diets (e.g., diabetics). However, scientists disagree about the relationships between sweeteners and lymphomas, leukemias, cancers of the bladder and brain, chronic fatigue syndrome, Parkinson's disease, Alzheimer's disease, multiple sclerosis, autism, and systemic lupus. Recently these substances have received increased attention due to their effects on glucose regulation. Occupational health nurses need accurate and timely information to counsel individuals regarding the use of these substances. This article provides an overview of types of artificial sweeteners, sweetener history, chemical structure, biological fate, physiological effects, published animal and human studies, and current standards and regulations.
Artificial sweeteners and the risk of gastric, pancreatic, and endometrial cancers in Italy.
Bosetti C, Gallus S, Talamini R, Montella M, Franceschi S, Negri E, La Vecchia C.
Istituto di Ricerche Farmacologiche Mario Negri, Milan, Italy. bosetti@marionegri.it
Abstract
BACKGROUND: The role of sweeteners on cancer risk has been widely debated over the last few decades. To provide additional information on saccharin and other artificial or low-calorie sweeteners (mainly aspartame), we updated the analysis of an integrated network of case-control studies conducted in Italy between 1991 and 2004 including data on cancers of the stomach, pancreas, and endometrium. PATIENTS AND METHODS: Cases were 230 patients with incident, histologically confirmed cancers of the stomach and 547 corresponding controls, 326 of the pancreas and 652 controls, and 454 of the endometrium and 908 controls. All controls were patients admitted to the same hospitals as cases for acute, non-neoplastic disorders. Odds ratios (OR) and corresponding confidence intervals (CI) were derived by unconditional logistic regression models. RESULTS: After allowance for various confounding factors, ORs for ever users of sweeteners versus nonusers were 0.80 (95% CI, 0.45-1.43) for gastric cancer, 0.62 (95% CI, 0.37-1.04) for pancreatic cancer, and 0.96 (95% CI, 0.67-1.40) for endometrial cancer. Corresponding ORs for saccharin were 0.65, 0.19, and 0.71, and for other sweeteners were 0.86, 1.16, and 1.07, respectively, for the three cancer sites. CONCLUSIONS: The present study adds further evidence on the absence of an adverse effect of low-calorie sweetener (including aspartame) consumption on the risk of common neoplams in the Italian population.
Aspartam wird abgebaut zu Phenylalanin, Asparaginsäure und Methanol.
Methanolanteil geringer, als z.b. in Apfelsaft.
Was soll daran jetzt schädlich sein, außer du leidest unter einer Phenyketonurie?
Das Methanol in Früchten ist aber an Pektin gebunden.
Der menschliche Organismus hat nicht das Enzym um das Methanol vom Pektin abzuspalten.
Das heißt, es verlässt den Organismus unverändert.
Der Organismus ist also nicht Methanol ausgesetzt, da es an Pektin gebunden ist.
Das ist der große Unterschied zum Aspartam.
Hier wird der Organismus freiem Methanol ausgesetzt.
Zitieren
Zitat von Mission Control
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